AbstractPDF
Abstract
Diabetes mellitus is a common cause of hyporeninaemic
hypoaldosteronism that might result in significant
hyperkalaemia. We describe a patient with diabetic
nephropathy and proteinuria who developed a remarkable hyperkalaemia on treatment with an angiotensin-receptor blocker. The management of hyperkalaemia and the pathophysiological background of hyporeninaemic hypoaldosteronism are discussed.
